Equipment
Over the years, Badger Iron Works has invested in machinery and equipment to meet the demands of a fast moving market. This list is a sampling of the current inventory of equipment to meet those needs.
Molding Machines
- Hunter HMP-32 30×32 automatic molding machine
- Hunter HMP-20 20×24 automatic molding machine
- Beardsley & Piper BSM-2016 16×20 Match-blomatic
- Osborn Rotolift and jolt-squeezer
- Fully automated Hartley PLC controlled sand system
No-bake Molding Line
- Castec high speed continuous mixer
- Gudgeon Thermfire sand reclaimation system (we reclaim 100% of our no-bake sand)
Core Making Equipment
- Beardsley & Piper ABC-6 and ABC-25 core machines
- Beardsley & Piper and Dependable shell core machines
- CO2
Melting
- 2 Brown Boveri (BBC) 5,000 lb. capacity induction melters
Testing Equipment
- Jarrell-Ash spectrometer
- Tinius Olsen “Super L” tensile testing machine
- Brinell hardness testing
- Olympus-photo microscope for microscopic examination
- Finite Solutions SolidCast® – solidification simulation computer system
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or NIC GFP in cells that were pretreated with Authenitc Blue Scott Hartnell Jersey siRNA to Akt1 or a scrambled control for 48 Cultures were continued with complete medium (CM) or buffered saline as described in Figure 2a. Apoptotic nuclear damage was assessed in GFP cells after 6 inset: levels of Akt1 protein in cells treated with siRNA. Authenitc David Clarkson Jersey (c) HeLa cells transfected with GFP or NIC GFP with or without DN Akt as indicated were cultured in CM or buffered saline. Apoptotic nuclear damage was assessed in GFP cells after 6 data represent mean from a minimum of three separate experiments. In panels b and c, the data are normalized to the CM condition Full figure and legend (95K) NIC GFP mediated inhibition of apoptosis was abrogated in cells where Akt1 protein was depleted using siRNA (Figure 3b), or following the cotransfection of a DN (defective ATP binding mutant) construct of Akt (DN Akt; Figure 3c), positioning Akt as an intermediate in the pathway activated by Notch. As Notch lacks intrinsic kinase activity, we speculated that